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Rotatory Vertebral Artery Syndrome in Foramen Magnum Stenosis
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Ileok Jung, Jin-Man Jung, Moon Ho Park
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Res Vestib Sci. 2018;17(4):167-169. Published online December 21, 2018
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DOI: https://doi.org/10.21790/rvs.2018.17.4.167
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Abstract
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- Rotatory vertebral artery syndrome (RVAS) is characterized by recurrent attacks of vertigo, nystagmus, and syncope induced by compression of the vertebral artery during head rotation. A 60-year-old man with atlas vertebrae fracture presented recurrent attacks of positional vertigo. Left-beat, upbeat and count clock-wise torsional nystagmus occurred after lying down and bilateral head roll (HR) showing no latency or fatigue. Magnetic resonance imaging revealed foramen magnum stenosis (FMS) and dominancy of right vertebral artery (VA). The flow of the right VA on transcranial Doppler decreased significantly during left HR. The slower the velocity was, the more the nystagmus was aggravated. RVAS can be evoked by FMS causing compression of the VA. And the nystagmus might be aggravated according to the blood flow insufficiency.
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- A Case of Rotational Vertebral Artery Syndrome after Vertebral Artery Dissection
Song Jae Lee, Ha Young Byun, Seung Hwan Lee, Jae Ho Chung Korean Journal of Otorhinolaryngology-Head and Nec.2020; 63(5): 228. CrossRef
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Rotatory Vertebral Artery Syndrome in Foramen Magnum Stenosis
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Ileok Jung, Jin-Man Jung, Moon-Ho Park
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Received November 21, 2018 Accepted December 5, 2018 Published online December 5, 2018
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[Accepted]
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Abstract
- Reversible vertebral artery syndrome (RVAS) is characterized by recurrent attacks of vertigo, nystagmus, and syncope induced by compression of the vertebral artery during head rotation. A 60-year old man with atlas vertebrae fracture. presented recurrent attacks of positional vertigo. Left-beat, upbeat and count clock-wise torsional nystagmus occurred after lying down and bilateral head roll (HR) showing no latency or fatigue. MRI revealed foramen magnum stenosis (FMS) and dominancy of right vertebral artery (VA). The flow of the right VA on trans-cranial Doppler decreased significantly during left HR. The slower the velocity was, the more the nystagmus was aggravated. RVAS can be evoked by FMS causing compression of the VA. And the nystagmus might be aggravated according to the blood flow insufficiency.
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Vestibular Paroxysmia and Hemifacial Spasm by Vascular Compression
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Ileok Jung, Do-Young Kwon
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Res Vestib Sci. 2017;16(4):171-173. Published online December 15, 2017
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DOI: https://doi.org/10.21790/rvs.2017.16.4.171
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Abstract
PDFSupplementary Material
- Vestibular Paroxysmia and facial spasm may be caused by vascular compression of the vestibular and facial root entry zone. We report a case of paroxysmal nystagmus accompanied by facial spasm and which is well visualized by three-dimensional reconstruction images. The 3-dimensional reconstruction image supports the view that vestibular paroxysmia may occur with hemifacial spasm simultaneously due to vascular compression.
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Vestibular paroxysmia and hemifacial spasm by vascular compression
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Ileok Jung, Do-Young Kwon
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Received September 4, 2017 Accepted November 21, 2017 Published online November 21, 2017
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[Accepted]
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Abstract
- Vestibular Paroxysmia and facial spasm may be caused by vascular compression of the vestibular and facial root entry zone. We report a case of paroxysmal nystagmus accompanied by facial spasm and which is well visualized by three-dimensional reconstruction images. The 3-dimensional reconstruction image supports the view that vestibular paroxysmia may occur with hemifacial spasm simultaneously due to vascular compression.
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Saccadic oscillations as a possible indicator of dizziness due to choline esterase inhibitors: an observational study with video-oculography
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Ileok Jung, Moon-Ho Park, Ji-Soo Kim
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Received August 7, 2024 Accepted August 20, 2024 Published online August 26, 2024
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DOI: https://doi.org/10.21790/rvs.2024.015
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Abstract
- Introduction: Cholinesterase inhibitors (ChEIs) are widely used to treat mild to moderate Alzheimer disease and vascular dementia. Even though dizziness due to medication of ChEIs has been ascribed to adverse effects on the cardiovascular or central nervous system, the mechanisms remain unclear and objective indicators are not available.
Methods We recorded the eye movements using video-oculography in three patients who developed dizziness and unsteadiness after initiation or increment of ChEI, donepezil (Aricept).
Results The patients showed frequent saccadic oscillations that improved after discontinuation of ChEI.
Conclusions Frequent saccadic oscillations may be an indicator of dizziness in patients taking ChEIs.
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